Figure 9From: Pepsin-pancreatin protein hydrolysates from extruded amaranth inhibit markers of atherosclerosis in LPS-induced THP-1 macrophages-like human cells by reducing expression of proteins in LOX-1 signaling pathwayPotential mechanism of action of the EAH by reducing expression of proteins associated with LOX-1 signaling pathway. The red arrows indicate the modulation obtained in this study, for each marker, due to EAH treatment. Numbers 1, 2, 3, 4 and 5 indicate the stages of the atherosclerosis process.Back to article page